ISLAMABAD: The research study established that men start developing an increased heart disease risk from the age of 30 until their death.
The research shows that men start experiencing heart disease risk between the ages of 30 and 40 because their risk develops seven years earlier than it does for women.
The Northwestern University study, which examined 5,100 subjects for three decades, tracked their health status from 1986 until 2020. The research study established that men should begin heart disease screenings after their 30th birthday to decrease heart disease death rates.
Researchers discovered that men aged 50 face a heart disease risk that exceeds that of women at the same age by 5 percent. The risk of heart failure and heart attack increases at an earlier age for men, while both genders experience similar stroke risks.
The study discovered that smoking and high blood pressure and diabetes create heart disease risk factors that affect men more than women.
The findings show that men reach 35 years old which marks the beginning of their increasing heart disease risk that continues throughout their middle-aged years.
The findings appeared in the Journal of the American Heart Association.
Sneezing or struggling? Scientists reveal why cold hits differently
Researchers have established the reasons that some individuals experience severe cold symptoms while others only develop mild sneezing.
An international news agency study shows that the body develops two different defense mechanisms which start the initial protection process inside the nasal cavity. The researchers discovered that the nasal lining functions as a protective barrier that prevents cold viruses from entering at their initial point of contact.
The study describes how nasal cells function as the initial defense mechanism that protects the body from rhinovirus viruses which causes the common cold. The infection will stay under control until severe symptoms develop if the cells begin reacting to the virus entry.
Researchers found that proteins interferons control the initial defense response. The proteins activate infected cells while they send protective signals to nearby uninfected cells which need to defend themselves against the virus. The virus spreads rapidly when the interferon response experiences a delay, which then results in excessive mucus production, breathing difficulties and inflammation.
Researchers at Yale School of Medicine developed a laboratory model that mimics human nasal lining by using nasal stem cells. The model included cells that produce mucus and cilia which are found in the nasal cavity. The researchers found that when they obstructed virus detection mechanisms in the model, the rhinovirus spread throughout the system and destroyed all of the cells in most instances.
The lead researcher, Dr. Bao Wang explained that his study found a rapid response of interferon will control rhinovirus infections without needing assistance from other immune system elements.
The senior researcher, Dr. Ellen Foxman demonstrated through her findings that human body responses to viruses determine illness severity more than the viruses themselves.
The scientists identified two limitations of laboratory models, which fail to accurately depict human physiology because they lack essential environmental elements and complete immune system functions.
The researchers stated that additional studies must be conducted to achieve a comprehensive understanding of common colds and all other respiratory infections.
